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Silicosis Continues To Be Deadly Health Hazard; New Risks


Silicosis is a preventable occupational lung disease caused by the inhalation of respirable (breathable) crystalline silica dust. Silicosis can progress to respiratory failure and death. No effective specific treatment for silicosis is available; patients are provided supportive care, and some patients may be considered for lung transplantation. Chronic silicosis can develop or progress even after occupational exposure has ceased.

 

The number of deaths from silicosis declined from 1,065 in 1968 to 165 in 2004. Hazardous occupational exposures to silica dust have long been known to occur in a variety of industrial operations, including mining, quarrying, sandblasting, rock drilling, road construction, pottery making, stone masonry, and tunneling operations. Recently, hazardous silica exposures have been newly documented during hydraulic fracturing of gas and oil wells and during fabrication and installation of engineered stone countertops. Because of new operations and tasks placing workers at risk for silicosis, efforts to limit workplace exposure to crystalline silica need to be maintained.

 

To describe temporal trends in silicosis mortality in the United States, the CDC analyzed annual multiple cause-of-death data for 2001to 2010 for decedents 15-years-old or older. From 2001 to 2010, a total of 1,437 decedents had silicosis coded as an underlying or contributing cause of death. The annual number of silicosis deaths declined from 164 (death rate = 0.74 per 1 million population) in 2001 to 101 (0.39 per 1 million) in 2010. Of these, 28 (1.9%) were aged 15–44 years, 1,370 (95.3%) were males, and 1,236 (86.0%) were whites. The overall age-adjusted silicosis death rate for blacks (0.87 per 1 million) was significantly higher than the rate for whites (0.59) and other races (0.16). The age-adjusted silicosis death rate for males (1.39 per 1 million) was significantly higher than the rate for females.

 

A statistically significant decline in silicosis death rates was observed during 2001–2010. However, silicosis deaths still occurred among persons aged 15–44 years. Of 28 decedents aged 15–44 years, the youngest was aged 19 years. This would be consistent with the decedent developing acute silicosis after an extremely high exposure to breathable crystalline silica. These findings indicate the importance of educating at-risk workers and their employers regarding the dangers of exposure to respirable crystalline silica in the workplace. The disparities by sex and by race reflect differences in the composition of the workforces in the industries and occupations placing workers at risk for exposure to crystalline silica dust.

 

Approximately two million U.S. workers remain potentially exposed to respirable crystalline silica. Occupational exposures to dust containing crystalline silica have long been known to occur in mining, quarrying, sandblasting, pottery making, rock drilling, road construction, stone masonry, and tunneling operations. Despite enforceable limits on worker exposure to respirable crystalline silica, substantial overexposures continue to occur in the United States. Moreover, new job tasks that place workers at risk for silicosis continue to emerge.

 

In 2004, occupational disease surveillance programs in Michigan, New Jersey, Massachusetts, New York, and Ohio reported nine confirmed cases of silicosis among technicians who performed sandblasting in dental laboratories. In 2013, there were approximately 37,000 dental laboratory technicians in the United States.

 

In a 2012 report from Israel, a 2014 report from Spain, and a 2015 report from the United States, silicosis has been documented among workers exposed to respirable crystalline silica dust during the fabrication and installation of quartz-containing engineered stone products used primarily for kitchen and bathroom countertops. Although not in the United States, silicosis cases have been reported in other occupational settings, including among denim sandblasters.

 

A 2013 report documented high levels of exposure to respirable crystalline silica during hydraulic fracturing of gas and oil wells. Moreover, a 2010 study reported an excess risk for silicosis in coal miners that was associated with silica as a component of coal mine dust formed during drilling, crushing, and loading of mine material. In 2013, there were approximately 204,000 oil and gas extraction industry workers and approximately 80,000 coal mining industry workers in the United States.

 

In 1999, the Council of State and Territorial Epidemiologists made silicosis a nationally notifiable condition. In addition, because current permissible exposure limits for breathable crystalline silica do not adequately protect workers, the Occupational Safety and Health Administration (OSHA) has proposed amending the current standards. One of the proposed changes is a lower permissible exposure limit.

 

The findings in this report are subject to at least three limitations. First, silicosis deaths were not validated by medical records or follow-up with health care providers, thus findings might be subject to misclassification. Second, no individual work history is reported on death certificates. Therefore, it was not possible to identify those industries and occupations where the decedents' exposures to crystalline silica occurred. Finally, inhalation of crystalline silica can cause diseases other than silicosis, such as lung cancer and chronic obstructive pulmonary disease, which are not considered in this analysis.

 

Effective silicosis prevention strategies for employers recommended by OSHA and the CDC's National Institute for Occupational Safety and Health are available. Comprehensive silicosis prevention programs include substituting less hazardous noncrystalline silica alternatives when possible, implementing engineering controls (e.g., blasting cabinets; local exhaust ventilation; not using compressed air for cleaning surfaces; using water sprays to control airborne dust; and using surface wetting to prevent dust from becoming airborne when cutting, drilling, grinding, etc.), administrative and work practice controls, personal respiratory protective equipment, medical monitoring of exposed workers, and worker training.

 

Because of the serious health and socioeconomic consequences of silicosis, new operations and tasks placing workers at risk for silicosis, and the continuing occurrence of silicosis deaths, efforts to limit workplace exposure to crystalline silica need to be maintained. At the same time, because of the sometimes long latency of silicosis, with cases diagnosed years after exposure and often in retirement, ongoing silicosis surveillance is needed to track its prevalence in the United States.

 

See the CDC Report

 

 

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